File:Synapse diagram which shows a recreation of William Utermohlen's portraits.jpg
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Summary
| Description | "Glial purinergic signaling under homeostatic conditions and in proteinopathies. Under healthy conditions (Left), cell-specific glial purinergic signaling contributes to fine-tuning of synapse function and thus, normal cognitive abilities. In proteinopathies (Right), accumulation of protein aggregates leads to glial phenotype changes that are associated with altered expression of purinergic signaling components and altered ATP and adenosine (Ado) levels. This impacts synaptic function and ultimately results in synapse loss, contributing to impaired cognition. Two self-portraits are shown to illustrate the cognitive status in health and disease, similar to the famous work of William Utermohlen, who continued to create self-portraits after being diagnosed with AD." |
| Date | Publication date of article: 14 May 2021 |
| Source | Glial Purinergic Signaling in Neurodegeneration |
| Author | The authors of the article are Marie J. Pietrowski, Amr Ahmed Gabr, Stanislav Kozlov, David Blum, Annett Halle, and Kevin Carvalho. The journal that published this article is Frontiers in Neurology |
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| Date/Time | Thumbnail | Dimensions | User | Comment | |
|---|---|---|---|---|---|
| current | 15:29, 31 May 2022 | | 1,084 × 682 (455 KB) | Realmaxxver | {{Information |Description="Glial purinergic signaling under homeostatic conditions and in proteinopathies. Under healthy conditions (Left), cell-specific glial purinergic signaling contributes to fine-tuning of synapse function and thus, normal cognitive abilities. In proteinopathies (Right), accumulation of protein aggregates leads to glial phenotype changes that are associated with altered expression of purinergic signaling components and altered ATP and adenosine (Ado) levels. This impact... |
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